By American Academy of Ophthalmology, Rod Foroozan MD
Offers a symptom-driven method of the prognosis and remedy of significant neuro-ophthalmic stipulations. With the point of interest at the sufferer, this e-book emphasizes exam and applicable adjunctive reviews, together with a dialogue of diagnostic imaging modalities, and leads the reader in the course of the occasionally sophisticated manifestations of neuro-ophthalmic sickness to anatomical localization of lesions and definitive analysis. an outline of the anatomy of visible pathways is followed by way of many illustrations.
Upon crowning glory of part five, readers might be capable to:
- Describe a symptom-driven method of sufferers with universal neuro-ophthalmic court cases with a purpose to formulate a suitable differential diagnosis
- opt for the main applicable assessments and imaging, according to symptomatology, to diagnose and deal with neuro-ophthalmic issues in an economical manner
- determine eye stream issues and the ocular motor process
Read or Download 2014-2015 Basic and Clinical Science Course (BCSC): Section 5: Neuro-Ophthalmology PDF
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Extra resources for 2014-2015 Basic and Clinical Science Course (BCSC): Section 5: Neuro-Ophthalmology
Burst information is supplied from the PPRF that is directly adjacent to the CN VI nucleus and MLF. The burst cells are normally inhibited by omnipause neurbris located in the RIP. Saccades are thought to be initiated by supranuclear inhibition of the omnipause cells, which allows burst cell impulses to activate the horizontal and vertical gaze centers (Fig 1-29). To produce horizontal movement of both eyes, a signal to increase firing must be distributed to the ipsilateral lateral rectus and the contralateral medial rectus muscles.
These collaterals may be interrupted if the conjunctiva and Tenon capsule are removed from the limbus during ocular surgery. Distal to the origin of the OphA, the intradural supraclinoid ICA gives off the anterior choroidal artery (AChoA) and anastomoses with the proximal posterior cerebral artery (PCA) through the posterior communicating artery (PCoA). The AChoA supplies blood to the optic tract and distally to the lateral geniculate nucleus (LGN) (Fig 1-11). Injury to the AChoA can produce the optic tract syndrome, a disorder consisting of contralateral homonymous hemianopia, contralateral band atrophy of the optic disc, and a contralateral ....
Injury to fibers within the radiations produces a hornan mous h,emianopia, a conhalateral visual field defect that respects the vertical midline. If ~~:corresponding fibers froin the 2 eyes are in close proximity, the field defect is identical i n eacli eye (congruou ). Congruou field defects occur with lesions involving the calcarine cortex. More anterior involvement often p roduces incongruous field defects, suggesting that the corresponding fibers lie farther apart more anteriorly in the visual pathways.