Blood-Spinal Cord and Brain Barriers in Health and Disease by Hari Shanker Sharma

By Hari Shanker Sharma

  • '''Blood-Spinal twine and mind obstacles in health and wellbeing and disorder' is a latest and intensely informative quantity that are meant to be a prepared resource of reference for all researchers and clinicians enthusiastic about the CNS in well-being and disease.'' --Cerebrospinal Fluid examine (June 2005)

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Butcher, E. , and Michie, S. A. (1998b). Adhesion molecule phenotype of T lymphocytes in inflamed CNS. J. Neuroimmunol. 84, 92–104. , Hartung, H. , and Toyka, K. V. (2000). Animal models for autoimmune demyelinating disorders of the nervous system. Mol. Med. Today 6, 88–91. , and Madri, J. A. (2000). Distinct roles for matrix metalloproteinase-2 and α4-integrin in autoimmune T cell extravasation and residency in brain parenchyma during experimental autoimmune encephalomyelitis. J. Neuroimmunol.

Cserr, H. , and Knopf, P. M. (1992). Cervical lymphatics, the bloodbrain barrier and the immunoreactivity of the brain: A new view. Immunol. Today 13, 507–512. , and Schulz, M. (1997). E- and P-selectin are not involved in the recruitment of inflammatory cells across the blood-brain barrier in experimental autoimmune encephalomyelitis. Blood 90, 4459–4472. , and Hoch, G. (1998a). The development of experimental autoimmune encephalomyelitis in the mouse requires alpha4-integrin but not alpha4beta7-integrin.

And Butcher, E. C. (1995). α4 integrins mediate lymphocyte attachment and rolling under physiologic flow. Cell 80, 413–422. , Weissman, I. , and Veromaa, T. (1999). Antibodies to CD44 and integrin alpha4, but not L-selectin, prevent central nervous system inflammation and experimental encephalomyelitis by blocking secondary leukocyte recruitment. Proc. Natl. Acad. Sci. USA 96, 6896–6901. Butcher, E. C. (1991). Leukocyte-endothelial cell recognition: Three (or more) steps to specificity and diversity.

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