Cardiology Drug Update, An Issue of Cardiology Clinics by JoAnne Foody MD FACC FAHA

By JoAnne Foody MD FACC FAHA

New cardiac medications are being built at a fast speed. numerous new medicinal drugs and sessions of substances are reviewed during this factor, which can help you cardiologists preserve up to date with the newest drug details.

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74 Furthermore, HDL from torcetrapib-treated subjects (120 mg daily) was also shown to increase macrophage cholesterol efflux primarily through ABCG1,75 possibly enhanced by increased content of LCAT and apoE. Thus the concept that CETP inhibition impairs cholesterol efflux is not supported by experimental data; however, promotion of cholesterol efflux may require adequate increases in HDL-c levels. The issue of whether CETP inhibition could impair RCT by slowing the return of HDL-derived cholesterol to the liver is more complex.

48. Villamil A, Chysant SG, Calhoun D, et al. Renin inhibition with aliskiren provides additive antihypertensive efficacy when used in combination with hydrochlorothiazide. J Hypertens 2007;25:217–26. 49. Oparil S, Yarows SA, Patel S, et al. Efficacy and safety of combined use of aliskiren and valsartan in patients with hypertension: a randomized double-blind trial. Lancet 2007;370:221–9. 50. Sealey JE, Laragh JH. Aliskiren, the first renin inhibitor for treating hypertension: reactive renin secretion may limit its effectiveness.

Me´nard J, Boger RS, Moyse DM, et al. Dosedependent effects of the renin inhibitor zankiren HCl after a single oral dose in mildly sodium-depleted normotensive subjects. Circulation 1995;91:330–8. 26. Rongen GA, Lenders JW, Kleinbloesem CH, et al. Efficacy and tolerability of the renin inhibitor Ro 42-5892 in patients with hypertension. Clin Pharmacol Ther 1993;54:567–77. 27. Camenzind E, Nussberger J, Juillerat L, et al. Effect of the renin response during renin inhibition: oral Ro 42-5892 in normal humans.

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